intellesis trainable segmentation module (Carl Zeiss)
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Carl Zeiss
intellesis trainable segmentation module
Intellesis Trainable Segmentation Module, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 93/100, based on 21 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/intellesis+trainable+segmentation+module/ZEN+Module+Intellesis/10__3390_slash_biomedicines14102148-104-1-6
Average 93 stars, based on 21 article reviews
Intellesis Trainable Segmentation Module, supplied by Carl Zeiss, used in various techniques. Bioz Stars score: 93/100, based on 21 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/intellesis+trainable+segmentation+module/ZEN+Module+Intellesis/10__3390_slash_biomedicines14102148-104-1-6
Average 93 stars, based on 21 article reviews
intellesis trainable segmentation module - by Bioz Stars,
2026-11
93/100 stars
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Software:Article Title: Differential effects of Smad2 and Smad3 in regulation of macrophage phenotype and function in the infarcted myocardium. Article Snippet: TGF-βs regulate macrophage responses, by activating Smad2/3.. We have previously demonstrated that macrophage-specific Smad3 stimulates phagocytosis and mediates anti-inflammatory macrophage transition in the infarcted heart.. However, the role of macrophage Smad2 signaling in myocardial infarction remains unknown. Article Title: Diabetes Induces Cardiac Fibroblast Activation, Promoting a Matrix‐Preserving Nonmyofibroblast Phenotype, Without Stimulating Pericyte to Fibroblast Conversion Article Snippet: .. Using default algorithms of the Article Title: BAK contributes critically to necrosis and infarct generation during reperfused myocardial infarction. Article Snippet: At least seven cell death programs are activated during myocardial infarction (MI), but which are most important in causing heart damage is not understood.. Two of these programs are mitochondrial-dependent necrosis and apoptosis.. The canonical function of the pro-cell death BCL-2 family proteins BAX and BAK is to mediate permeabilization of the outer mitochondrial membrane during apoptosis allowing apoptogen release. Staining:Article Title: Differential effects of Smad2 and Smad3 in regulation of macrophage phenotype and function in the infarcted myocardium. Article Snippet: TGF-βs regulate macrophage responses, by activating Smad2/3.. We have previously demonstrated that macrophage-specific Smad3 stimulates phagocytosis and mediates anti-inflammatory macrophage transition in the infarcted heart.. However, the role of macrophage Smad2 signaling in myocardial infarction remains unknown. |